| For medical practitioners and osteopaths - Osteopathy Journal Articles Catalog. |
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| Insertion tendon osteopathy in the section of the cervical spine. Manz, A. (1957), Zentralbl Allg Pathol 97(1-2): 71-9. |
| Interview with Paule Lemoine, osteopath Lemoine, P. (1986), Rev Orthop Dento Faciale 20(1): 99-105. |
| Intestinal absorption of calcium: physiological values and results in osteopathy Kapitola, J., J. Hrba, et al. (1985), Vnitr Lek 31(5): 451-6. |
| Intestinal osteopathy following partial gastric resection Schafer, H. and C. Baerwald (1992), Dtsch Med Wochenschr 117(5): 177-80. Abstract: 26 years after a partial gastric resection (Billroth II) for recurrent gastric ulcer a 62-year-old man developed severe intestinal osteopathy. For three years he had increasing pain in the lower back and hip with a noticeable waddling gait. Serum concentration of calcium (2.0 mmol/l) and 25-hydroxy-vitamin D3 (38 mmol/l) were reduced, those of alkaline phosphatase (572 U/l) and parathormone (532 pg/ml) increased. Radiology demonstrated Looser's zones in the ribs and iliac crest. Osteodensitometry showed obviously diminished bone density. Iliac crest biopsy revealed signs of osteomalacia and secondary hyperparathyroidism. Within three months of starting oral vitamin D3 and calcium the symptoms had definitely receded and serum concentrations of calcium and alkaline phosphatase had become normal (2.4 mmol/l and 156 U/l, respectively). Osteopathic symptoms are often the expression of an abnormal calcium/phosphate metabolism. The cause often lies in the gastrointestinal tract; not rarely it is a late complication of a gastrojejunostomy. |
| Intestinal osteopathy. Nitschke, U. and I. Giegler (1961), Klin Wochenschr 39: 733-9. |
| Intra-familial coexistence of genotypic condensing osteopathy and chronic psychosis. Study of HLA genotypes Goudemand, M., C. Goudemand-Joubert, et al. (1977), Nouv Presse Med 6(37): 3434. |
| Is the hyperostotic spondylosis deformans a diabetic osteopathy? Schoen, D., M. Eggstein, et al. (1969), Fortschr Geb Rontgenstr Nuklearmed 110(4): 524-39. |
| Kinesiological study of osteopathy, chiropractic and bone-setting. Nagera, J. M. and A. M. Sanchez (1950), Dia Med 22(29): 1005-7. |
| Laboratory diagnosis of metabolic osteopathy Schmidt-Gayk, H. (1996), Med Klin (Munich) 91 Suppl 1: 38-9. |
| Leukemic osteopathy and rheumatoid form of leukemia. Nicola, P. (1957), Minerva Pediatr 9(40): 1045-59. |
| Leukoycte chromosome study in osteopetrosis and in condensing osteopathy (Steida's islet type) Galimberti, A., G. Riggio, et al. (1965), Arch Ortop 78(6): 417-22. |
| Levator ani syndrome, functional intestinal disorders and articular abnormalities of the pelvis, the place of osteopathic treatment Riot, F. M., P. Goudet, et al. (2004), Presse Med 33(13): 852-7. Abstract: OBJECTIVES: Various treatments of the Levator ani syndrome (LVAS) have been proposed. None of them alleviate symptoms in more than 20% of the cases. A new approach combining massages of the coccygeus muscle with physical treatment of frequently associated pelvic joint disorders was assessed. The outcome of the Irritable Bowel Syndrome (IBS) that is frequently associated was also studied. METHODS: One hundred and one patients (76 females and 25 males, mean age: 54 years) with a diagnosis of LVAS were studied prospectively over one year. Massages were given with a patient lying on the left side. Physical treatment of the pelvic joint disorders was given at the end of each massage session. RESULTS: Forty-seven patients (46.5%) suffered both from LVAS and IBS. A mean of less than 2 sessions of treatment was necessary. Sixty-nine per cent of the patients were LVAS-free at 6 months (p<0.0001) and 10% were improved. At 12 months, 62% were still free of symptoms and 10% improved (p=0.37). A comparable trend according to the Rome II criteria was found in the IBS-patient group (53% IBS free initially, 78% at 6 months p=0.00001, 72% at 12 months p=1). There was a significant correlation between the favorable outcome of IBS and the LVAS at 6 and 12 months. All IBS-free patients were LVAS-free at 6 months. CONCLUSION: The LVAS may be cured or alleviated in 72% of the cases at 12 months with one to 2 sessions. This new comprehensive treatment suggests the role of a muscular and osteoligamentary etiology in the LVAS. Since most of IBS benefitted from such treament, it is logical to suspect a mutual pathogeny and to screen for LVAS in all such patients. |
| Levels of vitamin A in the serum in dialyzed patients with renal osteopathy Sotornik, I., V. Novakova, et al. (1988), Cas Lek Cesk 127(33): 1037-42. |
| Long term CaCO3 treatment of chronic hemodialysis patients--an attempt to prevent and treat aluminum osteopathy Matsubara, M., K. Totsune, et al. (1988), Nippon Jinzo Gakkai Shi 30(11): 1349-54. |
| Mammectomy in the rat and its endocrine effects; observation of a generalized osteopathy. Lacour, F., J. Lacour, et al. (1953), Bull Assoc Fr Etud Cancer 40(3): 283-301. |
| Management of renal osteopathy in patients with chronic dialysis Kiss, D. and F. P. Brunner (1996), Schweiz Med Wochenschr 126(42): 1792-8. Abstract: During the past decade considerable progress has been made in the field of calcium metabolism associated with chronic renal failure. The main factor influencing the pathophysiology of calcium homeostasis is its dependence on parathyroid hormone and active vitamin D metabolites. Among several new diagnostic tools, and to establish the diagnosis of renal osteodystrophy, only the measurement of i-PTH activity has any clinical relevance. The main goal in treating renal osteodystrophy is the lowering of PTH activity by correcting hyperphosphatemia and administration of calcitriol given either orally or intravenously. Furthermore, the need to lower the calcium concentration in the dialysate during calcitriol treatment is demonstrated. The clinical significance of adynamic osteodystrophy is still unclear. |
| Manual therapy, chiropractic, osteopathy. From alternative therapy to medicine Ylinen, J., J. Piispanen, et al. (1996), Duodecim 112(14): 1264-73. |
| Marie-Bamberger hypertrophic osteopathy. Brezina, Z. (1957), Cesk Pediatr 12(10): 925-7. |
| Metabolic indicators of the causes and status of diabetic osteopathy Stepan, J., T. Havranek, et al. (1982), Vnitr Lek 28(6): 577-86. |
| Metabolic osteopathy (author's transl) Pacovsky, V. (1980), Cas Lek Cesk 119(6): 161-6. |
| Microalbuminuria as a risk factor for diabetic osteopathy in patients with IDDM and renal sufficiency Goliat, E., W. Marusza, et al. (1998), Pol Arch Med Wewn 100(2): 111-8. Abstract: Disturbances in bone marrow vascularisation can be one of the causes of diabetic osteopathy. The aim of the study was to answers the question if microalbuminuria as a results of capillary injury can be a sign of bone mineralisation disorders in IDDM renal sufficient patients. We examined 60 IDDM patients (30 women without menstruation disturbances; 30 men; age 25-36 years old). All the observed subjects were divided into groups: I-30 normoalbuminuric patients (0-29 mg/24 h); II-30 microabuminuric patients (30-295 mg/24 h). Bone mineral density (BMD) of femoral neck, lumbar spine (L2-L4) and total body was measured by dual energy X-ray absorptiometry (DEXA, Lunar). The biochemical parameters of bone turnover were measured both in serum and urine as follows: osteocalcine, total hydroxyproline (HPR, HPR/Cr), total alkaline phosphatase (AP) with bone fraction, total calcium (Ca, Ca/Cr) and inorganic phosphor (P). Microalbuminuric patients presented more severe bone turnover disturbances, shown by differences in: BMD and Z-score for femoral neck (p < 0.05), serum HPR (p < 0.05), AP (p < 0.05), AP (p < 0.01) and its bone fraction (p < 0.05). We proved the presence of statistically significant correlation coefficients for albuminuria and some densytometric and biochemical bone parameeters. Our results suggest that microalbuminuria can indirectly indicate the dynamic of bone turnover derangement in IDDM course. They are present mostly in the femoral neck, which because of the vascularisation type is particularly susceptible to subalimentation in the diabetic microangiopathy course. |
| Mineral content of bone in long-standing diabetes: densitometry of "diabetic osteopathy" (author's transl) Ringe, J. D., F. Kuhlencordt, et al. (1976), Dtsch Med Wochenschr 101(8): 280-2. Abstract: Using 125I-photon absorption, skeletal mineral content was measured in 57 diabetics requiring insulin. Average age was 33 years, mean duration of diabetes 19 years. Abnormally reduced mineral content was detected in ten of 21 males and 11 of 36 women. This finding is interpreted as being a late complication of diabetes. Pathological fractures were not seen and there were very few complaints referable to the skeletal system. |
| Modern concepts of osteopathy. Bartelheimer, H. (1954), Dtsch Med J 5(13-14): 409-12. |
| Monostotic Condensing Osteopathy Of The Ribs. Rigoli, E. (1963), Riv Anat Patol Oncol 24: 1297-309. |
| More on the Camurati-Engelmann osteopathy. Cozzolino, A. (1959), Arch Ortop 72: 1422-7. |
| Morphogenesis of occupational fluoride osteopathy Razumov, V. V., O. A. Klitsenko, et al. (1997), Med Tr Prom Ekol(4): 18-23. Abstract: Fluor osteopathy, as the authors suppose, is a morphologic repetition of phylogenesis early stages in osteogenesis. Thus, osteosclerosis and osteoporosis demonstrated by X-ray should be considered as manifestation of bone fluorosis. Fluor-induced changes of bone tissue could not be adequately termed as "osteoporosis" and "osteosclerosis", so is defined as "fluor osteopathy". |
| Morphometric studies in early stages of renal osteopathy (author's transl) Hufnagl, H. D., M. Hausen, et al. (1974), Klin Wochenschr 52(22): 1070-7. |
| Neuralgiform lumbosciatic osteopathy with vertebral and paravertebral localization. Bertola, L. and A. Pedrocca (1953), Minerva Ortop 4(7-8): 215-8. |
| Neurogenic osteopathy Dinkel, L. (1972), Radiologe 12(3): 101-2. |
| New histological method for diagnosing renal osteopathy from a single decalcified specimen (author's transl) Povysil, C., V. Vanecek, et al. (1980), Cas Lek Cesk 119(11): 337-8. |
| New osteopathic law. Femmer, H. J. and Perl (1956), Med Monatsschr 10(5): 322-7. |
| New viewpoints on the therapy and prevention of renal osteopathy Kaiser, W. and E. Mampel (1977), Z Gesamte Inn Med 32(12): 254-9. Abstract: Dihydrotachysterol which was developed already 50 years ago is above all used in the substitution therapy of hypoparathyroidism. Its parathyreomimetic effectivity has led to an enlargement of the spectrum of indication during the last years; it allows the prophylactic use of dihydrotachysterol in chronic nephropathy in order to prevent by this means the risk of reactive hyperparathyroidism as well as its sequelae. On the basis of own experimental results the pathophysiologic prerequisites of this new form of treatment are discussed. |
| Non-familial ulcero-mutilating acro-osteopathy of the feet Eichhorn, K. and S. Schauder (1989), Hautarzt 40(5): 316-8. |
| Non-familial ulcero-mutilating acro-osteopathy of the foot (Bureau-Barriere syndrome) Fluckiger, R., P. H. Itin, et al. (1993), Schweiz Rundsch Med Prax 82(1): 28-30. Abstract: We report on a case of severe acro-osteopathia ulcero-mutilans complicated by lethal endocarditis. The clinical course is presented with emphasis on the typical signs and symptoms. Diagnosis and treatment require a multidisciplinary approach to such patients. |
| Non-rachitic hypophosphatemic osteopathy Borzani, M., L. Mauri, et al. (1983), Pediatr Med Chir 5(3): 99-102. Abstract: We have identified a patient with a condition called Hypophosphatemic Bone Disease (HBD). This disorder of phosphate metabolism is unrecorded by Dent in his final classification of metabolic bone diseases and has been described by Scriver C.R. and coll. Although the condition is in same ways analogous to X-Linked Hyophosphatemia (XLH), there are important differences between the two diseases. For example, there is selective impairment in the tubular reabsorption of phosphate in HBD but the defect is less severe and it is clearly different from that described in XLH. Clinical manifestations of HBD appear in infancy, but the dwarfism and the bone changes are less severe than in XLH at comparable concentrations of plasmatic phosphorus in the two diseases. While in both conditions there is osteomalacia of endostal trabecular bone, only in XLH is florid rickets present, affecting the epiphyses and compromising linear growth. The phosphaturic response to PTH infusion is abnormal in qualitative aspects, but it is present in HBD, and this differs considerably from that described in XLH. The treatment with oral load of phosphates and 1,25(OH)2D3 in every patient with HBD, and so in our patient, is accompanied by increase in serum phosphorus, with improved tubular reabsorption of phosphate anion and a fall of hydrossiprolinuria with bone healing; this combination of responses is not present in XLH. |
| Non-tropical sprue, a frequently unrecognized cause of high-grade generalized osteopathy Kruse, H. P., J. D. Ringe, et al. (1987), Dtsch Med Wochenschr 112(30): 1155-9. Abstract: Average interval from initial symptoms to diagnosis in 47 patients with non-tropical sprue was 8.3 years. Predominantly skeletal symptoms without typical gastro-intestinal symptoms occurred in about one third of cases, probably a reason for the delay in diagnosis. In more than 90% of cases there was the histological picture of osteopathy, osteomalacia with or without secondary hyperparathyroidism being about twice as frequent than osteoporosis. Biochemical and histological findings correlated well. In more than 80% of cases the mean mineral content of the radius bone was reduced by more than 30% of normal, while radiological findings were abnormal in only just half the cases. Even in the absence of diarrhoea, non-tropical sprue should be considered in aetiologically uncertain cases of generalised osteopathy. |
| Notes on para-articular osteopathy. Callewaert, R. and H. Claessens (1959), Belg Tijdschr Geneesk 15(9): 452-6. |
| Observation of late sequelae following osteopathy after dystrophy. Schoelzel, P. and R. Tascher (1959), Medizinische 41: 1929-32. |
| Observations on mandibular osteopathy in children - doubling time of the lesion Tokita, S., H. Tsujikawa, et al. (1982), Josai Shika Daigaku Kiyo 11(2): 300-7. |
| Occupational osteopathy of rug weavers. Apropos of 64 cases Chahidi, H. (1975), J Chir (Paris) 109(5-6): 633-8. Abstract: Carpet weaving carried out by young girls in abnormal positions for long periods, causes in the latter important bony deformities, aggravated by poor hygiene and diet. Prevention and treatment is, however, possible. |