| For medical practitioners and osteopaths - Osteopathy Journal Articles Catalog. |
|
|
|
|
||
|
|
||
|
|
| Next Page | Last Page |
| "Osteopathic" is winning edge for Ohio contract bidders Onusko, T. J. (1986), Osteopath Hosp Leadersh 30(1): 15, 22. |
| "Traditional osteopathy": an oxymoron? Findlay, T. (2000), J Am Osteopath Assoc 100(9): 545. |
| "Hazards of chiropractic and osteopathic treatment". Degenring, F. W. (1961), Med Klin 56: 1755-6. |
| 2 Cases Of Ankylosing Spondylitis With Distinct Osteopathic Components. Corbella, E. G. and L. Muzio (1964), Reumatismo 16: 265-9. |
| 3 cases of Paget's familial deforming osteopathy. Etiopathogenetic, clinical and therapeutic aspects Falchi, G. (1966), Clin Ter 37(4): 293-324. |
| A case of congenital syphilis with osteopathy and meningitis Venezia, A., A. Cosma, et al. (1976), Minerva Pediatr 28(34): 2087-93. |
| A case of grave osteopathy during hypocythemic leukosis. Dulbecco, A. and M. Rossi (1960), Radiol Med (Torino) 46: 299-306. |
| A case of hyperostotic and sclerosing osteopathy. Schneider, M. (1959), Strasb Med 10(2): 104-11. |
| A case of osteopathy disseminated condensing. Grepl, J. (1954), Bratisl Lek Listy 9(24): 562-3. |
| A case of osteopathy in caisson disease; radiological aspects. Chaumont, A. J. and A. M. Blittersdorf (1953), Med Usine Rev Hyg Ind Mal Prof 15(4): 198-200. |
| A case of osteopathy Broussin, J. and J. P. Basseau (1972), Bord Med 5(6): 713-4 passim. |
| A case of rare osteopathy: Ollier's disease. Santagada, A. (1954), Arch Radiol 29(1): 117-25. |
| A case of renal osteopathy in the adult. Raynaud, R., M. Brochier, et al. (1958), Alger Medicale 62(9): 979-93. |
| A case of severe rarefaction osteopathy associated with tabes dorsalis Rivier, G., J. C. Gerster, et al. (1991), Rev Rhum Mal Osteoartic 58(11): 819-21. Abstract: The authors report the case of a 63 years old man who presented 13 years after a diagnosis of a tabes dorsalis, stress fractures of the lower limbs and Charcot's joints (knees). 7 years later, he had multiple stress fractures of the sacrum and pelvis and of the ribs following a minor trauma. Laboratory investigations revealed a major osteoporosis probably related to the neurochirurgical complications of the tabes dorsalis. |
| A condensing osteopathy sui generis: idiopathic osteomyelosclerosis in adults. Barone, L. and D. Barisone (1963), Radiol Med (Torino) 49: 16-33. |
| A contribution to endocrine osteopathy: Jaffe-Lichtenstein disease. Will, I. (1959), Munch Med Wochenschr 101: 2206-8. |
| A critical analysis of micromorphometry in metabolic osteopathy (author's transl) Ritz, E., B. Krempien, et al. (1974), Verh Dtsch Ges Pathol 58: 363-5. |
| A very unusual case of osteopathy during anticonvulsive treatment Decoulx, M., J. Lefebvre, et al. (1978), Lille Med 23(4): 234-8. |
| Acidosic tubular dysplasia, hyperaminoaciduria with malacic osteopathy and secondary hyperparathyroidism. Guideri, R., C. Gennari, et al. (1963), Arch Sci Med (Torino) 115: 63-103. |
| Acropachy in hemangiomatosis. (Comparative pathological anatomical, x-ray and clinical studies of acropachy and toxic hypertrophic osteopathy) Heidenblut, A., H. J. Kleinschmidt, et al. (1967), Fortschr Geb Rontgenstr Nuklearmed 107(4): 497-503. |
| Acute effect of porcine calcitonin in persons without osteopathy and patients with Paget's disease Courvoisier, B., Q. de Barros, et al. (1970), Schweiz Med Wochenschr 100(1): 26-32. |
| Alcoholic osteopathy Sarli, M., H. Plotkin, et al. (1994), Medicina (B Aires) 54(4): 363-70. Abstract: Alcohol intake is one of the multiple risk factors for developing osteoporosis. Alcohol has direct toxic effects on osteoblasts which determines lower osteocalcin levels at an early stage, and histomorphometric changes later on. Some authors found not only diminished bone formation in alcoholics, but also increased bone resorption. The effect of alcoholism on calciotropic hormones includes fall of PTH serum levels after an acute or moderate alcohol intake, causing transient hypoparathyroidism. In chronic alcoholism, serum levels of vitamin D and its metabolites are decreased independently of any liver disease, probably related to alcohol influence on enzymatic systems. The mineral homeostasis of alcoholics is affected: hypocalcemia is found in acute intoxication, with hypo or hypermagnesemia. In chronic alcoholism the serum calcium values tend to be normal. Alcohol intake causes multiple endocrine changes that lead to hypogonadism in both sexes. The stimulation of hypothalamus-hypophyseal-adrenal axis contributes to the alcoholic bone disease, because of the adverse effects of corticoids on bone. Caloric and protein malnutrition, in addition to a dissipated life style are additional risk factors for the development of osteoporosis. |
| Aluminum gels in chronic kidney failrue patients. A potential risk of encephalopathy and osteopathy Rottembourg, J., M. C. Jaudon, et al. (1980), Ann Med Interne (Paris) 131(2): 71-4. |
| Aluminum osteopathy in a patient hemodialysed for chronic renal insufficiency and treated by long term administration of sucralfate Level, C., V. de Precigout, et al. (1996), Rev Med Interne 17(4): 348-9. |
| Aluminum osteopathy Povysil, C., I. Sotornik, et al. (1990), Cesk Patol 26(2): 65-71. Abstract: Histochemical study of 155 bone biopsies from dialyzed patients revealed 43 cases with aluminium deposits in bone trabeculae localized along the mineralization line or in phagolysosomes of bone marrow macrophages. Aluminium was limited to macrophages in 5 cases, to mineralization line in 23 cases, and found in both localities in 15 patients. Patients with a positive finding mostly had osteomalacia (8 times) which was combined in some cases (35 times) with mild fibrous osteodystrophy. Authors failed to prove histochemically positive deposits in dialyzed patients with serious parathyroidism. Simultaneous deposits of aluminium and hemosiderin were found in the mineralization line in three patients. Bone deposition of aluminium seems to influence somehow the bone lesion but precise interpretation of the phenomenon has not been given. |
| Aluminum osteopathy--experience with therapy Sulkova, S. (1993), Vnitr Lek 39(5): 459-63. Abstract: The author discusses the problem of aluminium osteopathy as one of the clinical manifestations of aluminium intoxication in dialyzed patients. She describes the case-histories of three patients treated on account of aluminium intoxication with desferrioxamine. The disease was manifested by pain in the bones and joints and muscular weakness, in two patients also with symptoms of encephalopathy. The diagnosis was assessed only after a special examination of bone trepanobiopsy (staining with aurintricarboxyl acetic acid) and confirmed by assessment of the aluminium serum concentration. While treatment of the first patient in 1986 was not successful, in another two female patients it proved possible by combined treatment (desferrioxamine and haemofiltration treatment) to eliminate the aluminium from the organism and to improve the symptoms. At present no longer aluminium hydroxide is prescribed as a routine binding agent of phosphates in the digestive tract. Therefore cases of aluminium intoxication are rare. Nevertheless it is important to pay attention to the problem of aluminium accumulation as its early diagnosis facilitates treatment. In the author's opinion the method of choice for elimination of aluminium from the organism is one-line haemofiltration combined with desferrioxamine. |
| Aluminum-induced osteopathy--pathogenetic mechanisms Achenbach, H. (1989), Z Gesamte Inn Med 44(5): 133-7. Abstract: The causal connection of aluminium accumulation in the organism and of toxic aluminium effects on the bone metabolism is at present regarded as extensively ascertained. On the other hand, there is no final consent about the fact which exact pathomechanisms are the basis of the aluminium effects. Apart from the direct interaction of aluminium with osseous transformation processes influences of the aluminium load of the body on significant, modulating the bone turnover extraosseous factors could be proved. Discussions about the complex pathogenetic principles of aluminium-induced bone changes concentrate themselves upon inhibitory effects of aluminium in the process of mineralisation, upon immediate toxicity of aluminium on the bone cells, potential interactions between aluminium and changed NSD-function as well as the influence of the aluminium accumulation on the vitamin-D-balance. |
| Alveolar process in renal osteopathy Philipp, K. U. (1972), Zwr 81(15): 704-7. |
| An original case of cystic osteopathy in a child De Pra, M. and M. Guarino (1972), Minerva Pediatr 24(3): 106-12. |
| An unusual case of epiphysial rarefying osteopathy of the femur. Morisi, M. and O. Mantero (1955), Minerva Ortop 6(10): 407-11. |
| Anamnestic observations of renal osteopathy during hemodialysis using microradioscopy of the hand and histomorphometry of the iliac crest Schulz, W., W. Bosert, et al. (1977), Verh Dtsch Ges Inn Med 83: 1296-9. |
| Anatomo-pathological and clinico-radiological findings in thalassemic osteopathy. (Study of 3 cases) Beltrami, C. A. and M. Ortolani (1968), Riv Patol Clin Sper 9(2): 295-306. |
| Application of high resolution scanning electron microscopy in osteopathic surgery Tanaka, K. (1989), Nippon Seikeigeka Gakkai Zasshi 63(4): 370-6. |
| Apropos of hemophilic osteopathy; considerations on the indications to surgical therapy. Personal observation Campana, F. P. and R. Mammucari (1967), Ann Ital Chir 43(1): 15-26. |
| Arthropathy in primary oxalosis--crystal synovitis or osteopathy? (author's transl) Schmidt, K. L., H. W. Leber, et al. (1981), Dtsch Med Wochenschr 106(1): 19-22. |
| Aspects of familial dysplastic osteopathy. Crecelius, W. (1952), Fortschr Geb Rontgenstr Nuklearmed 76(2): 196-202. |
| Bilateral spontaneous fracture of the femoral neck in renal osteopathy; with a note on intestinal lavage in uremia. Dohrmann, R. (1953), Chirurg 24(10): 470-2. |
| Biochemical and x-ray changes in thyroid osteopathy Baleva, R., L. Dianov, et al. (1978), Vutr Boles 17(2): 79-86. Abstract: Moderately elevated calcemia, increased urine calcium excretion, normal values of phosphate clearance and tubular phosphate resorption, elevated alkaline phosphatase were found in 50 patients with clinically confirmed thyrotoxicosis. With calcium loading, an essential calcium elevation in serum was determined as well as a delayed restoration to the norm of the latter, providing grounds to admit an insufficiency of hypocalcemia leading systems, thyrocalcitonine resp. The characteristic features of the X-ray changes observed are described, especially of osteoporosis. The bone changes were established to be more frequent among females (64% versus 204 among males). They increase in parallel with age advancing, during and severity of the disease. The special X-grams for fine analysis of bone structure according to Meema-Heunk enable the confirmation of intracortical striation in metacarpal bones and phalanges in 56.7 per cent of the examined. |
| Biokinetics of osteotropic radiopharmaceuticals in metabolic osteopathy--comparison of 47Ca and 99mTc-methylene-diphosphonate (MDP) Knop, J. and R. Montz (1983), Nuklearmedizin 22(5): 226-31. Abstract: Plasma clearance and osseous accumulation rates of 99mTc methylene diphosphonate (MDP) in the lumbar spine were measured in 11 patients with osteoporosis (OP), 7 patients with hyperparathyroidism (HPT), 4 patients with osteomalacia (OM) and in 3 patients (N) who were studied to exclude metabolic bone disease. The findings in 19 patients were compared with the results of 47Ca kinetics. The plasma elimination of 99mTc-MDP during the first hour after application was normal in almost all the patients. The 99mTc-MDP accumulation rates in the lumbar spine were raised in patients with HPT and OM (p less than or equal to 0.01) and normal in patients with OP and N (p greater than 0.05). 47Ca kinetics were disturbed in most of the patients with metabolic bone disease, with minimal deviations in OP. The comparison of 99mTc-MDP accumulation rates with the results of 47Ca kinetics revealed significant correlations with exchangeable bone and soft tissue calcium. The correlations with total calcium turnover and calcium accretion were poor. Only half of the patients had equal results of 99mTc-MDP accumulation rates and bone calcium accretion rates. It is concluded that the 99mTc-MDP uptake by bone is an index of bone metabolism presumably of the organic matrix, whereas 47Ca kinetics represent the mineral metabolism of bone. |
| Bone changes induced by hormones in renal osteopathy. Studies on the macro- and microstructure of bones von Babo, H. and F. Heuck (1974), Radiologe 14(5): 225-31. |